Pathway Hepatitis

Hepatitis is an inflammation of the liver cells are expanded / spread, viral hepatitis is the most dominant species. Injury to the organ liver with inflammation can develop after the opening for a number of pharmacological and chemicals from inhalation, ingestion, or parenteral drug administration (IV). Toxin and Drug Induced Hepatitis is a result of the opening or the opening of hepatotoxin, such as industrial toxins, alcohol and medications that are used in medical therapy.

After the liver opened a number of agents such as viruses, the liver becomes enlarged and inflammation that occurs in the right upper quadrant pain and discomfort. As the advancement and continuation of the disease process, cell division, normal liver cells turned into a widespread inflammation, necrosis and regeneration of liver cells. Increased emphasis in the trajectory caused by viruses circulating in and mixes with the blood flow into the cleavage of hepatic tissues (liver cells). Edema of the liver bile ducts are present in intrahepatic causes yellowish tissue.

Specific data on the pathogenesis of hepatitis A, hepatitis C, hepatitis D and hepatitis E are very limited. The signs remind investigation on clinical manifestations of acute inflammation HBV determined by immunological responses from the client. Immune complex – Tissue damage indirectly allow for the manifestation of acute hepatitis B extrahepatik. Hepatitis B is believed to enter the circulation of immune stored in the walls of blood vessels and is active in the charging system. (Dusheiko, 1990). Clinical responses consisting of pain mixed with pain that occurs everywhere.

Phase or stage of healing of hepatitis is characterized by the activity of phagocytosis and the activity of enzymes, improvement of hepatic cells. If it does not really develop complications, most of the healing of the client as normal liver function after losing viral hepatitis. Complete regeneration usually occurs within two to three months.